Back and spine injuries end careers, destroy financial security, and permanently alter lives. A construction worker with lumbar disc herniations can no longer lift, bend, or perform the physical labor that supported his family. A nurse with chronic back pain cannot stand for 12-hour shifts. A truck driver with sciatica cannot sit for long hauls. These aren't temporary inconveniences - they're permanent disabilities requiring lifetime treatment and preventing return to previous employment. Yet insurance companies treat serious spine injuries as minor "back strains" deserving minimal compensation. Recovering fair damages requires attorneys who understand spinal anatomy, can prove injury severity with comprehensive medical evidence, and possess the trial experience to overcome aggressive insurance defense.
Understanding the Spine and Common Injury Types
The human spine consists of 33 vertebrae divided into five regions: cervical (neck, 7 vertebrae), thoracic (mid-back, 12 vertebrae), lumbar (lower back, 5 vertebrae), sacral (5 fused vertebrae forming the sacrum), and coccygeal (3-5 fused vertebrae forming the tailbone). Between most vertebrae sit intervertebral discs acting as shock absorbers. The spinal cord runs through the spinal canal formed by the vertebrae, with nerve roots branching off at each level.
Lumbar Spine - The Most Vulnerable Region
The lumbar spine bears the body's weight and experiences tremendous forces during lifting, bending, and twisting. This makes it the most common site of traumatic injury. The L4-L5 and L5-S1 disc levels experience the highest stress and most frequently herniate.
Lumbar injuries cause low back pain that can be localized or spread across the lower back and hips. When nerve roots are compressed, pain radiates down the legs following specific patterns. The sciatic nerve, formed from L4-S3 nerve roots, when compressed produces the characteristic leg pain called sciatica.
Thoracic Spine Injuries
The thoracic spine is more stable than cervical or lumbar regions due to rib cage attachment, making thoracic injuries less common but often more serious when they occur. Thoracic spine fractures, disc herniations at thoracic levels, and spinal cord injuries in the thoracic region can cause paralysis, respiratory problems, and severe disability.
Thoracic injuries typically result from high-energy trauma like high-speed collisions, falls from height, or direct impacts to the mid-back.
Herniated Discs - The Most Common Serious Spine Injury
Intervertebral discs consist of a tough outer ring (annulus fibrosus) surrounding a gel-like center (nucleus pulposus). When traumatic forces tear the annular fibers, nucleus material can herniate through the tear, creating a disc herniation.
How Disc Herniations Occur in Accidents
Multiple mechanisms cause traumatic disc herniations:
Axial loading: Vertical compression forces from falls landing on feet or buttocks, or roof crush in rollovers, compress discs between vertebrae. The disc's nucleus experiences sudden increased pressure, forcing it through any weak points or tears in the annulus.
Flexion injuries: Forward bending forces during frontal collisions or lifting injuries increase pressure on the anterior disc and stretch posterior annular fibers. The nucleus migrates posteriorly toward the weakened area, often herniating posterolaterally where nerve roots exit.
Extension injuries: Backward bending forces from rear-end impacts or falls landing on back create opposite stresses. While less likely to cause posterior herniations than flexion, extension can cause anterior herniations or damage the anterior annulus, predisposing to later herniation.
Rotation and torsion: Twisting motions during side-impact collisions or rotational falls create shearing forces exceeding the disc's tensile strength. These rotational injuries frequently cause large, complex herniations.
Types of Disc Herniations
Herniation Classifications
Disc bulge: Generalized extension of disc material beyond the vertebral margins. The annulus remains intact but stretched. Bulges may be asymptomatic or cause pain from nerve irritation. Insurance companies minimize bulges as "normal aging" despite evidence linking them to trauma.
Protrusion: Focal herniation where nucleus pulposus pushes through annular fibers but the outermost fibers remain intact, containing the herniated material. The herniation base is wider than the herniated material extending from it.
Extrusion: More severe herniation where nuclear material breaks through all annular layers. The herniated fragment's base is narrower than the fragment itself, creating a "mushroom" appearance. Extruded material can migrate up or down the spinal canal away from the disc space.
Sequestration: Most severe herniation type where disc fragment completely separates from the parent disc and becomes a free fragment in the spinal canal. Sequestered fragments can migrate significantly from the herniation site and are difficult to treat conservatively.
Direction matters: Posterior herniations extend backward toward the spinal canal and nerve roots, most likely to cause symptoms. Posterolateral herniations (most common) compress nerve roots exiting at that level. Central herniations can compress the spinal cord (in cervical/thoracic spine) or cauda equina (in lumbar spine). Foraminal herniations occur directly in the neural foramen where nerve roots exit.
Symptoms of Lumbar Disc Herniations
Herniation symptoms depend on location and degree of nerve compression:
Localized back pain: Often the first symptom. Pain from the disc itself and surrounding inflamed tissues. May be severe immediately or develop over days as inflammation progresses.
Radicular pain (sciatica): Sharp, burning, or electric shock-like pain following the compressed nerve's distribution. L4 radiculopathy causes pain down the anterior thigh to the knee. L5 radiculopathy produces pain down the lateral leg to the top of the foot and big toe. S1 radiculopathy causes pain down the posterior leg to the lateral foot and small toes.
Neurological deficits: Numbness and tingling in specific dermatome patterns corresponding to the compressed nerve root. Weakness in muscles supplied by the affected nerve - foot drop from L5 compression, weak ankle push-off from S1 compression. Reflex changes - diminished knee jerk with L4 involvement, absent ankle jerk with S1 compression.
Cauda equina syndrome: Surgical emergency occurring when massive central disc herniation compresses multiple nerve roots of the cauda equina. Symptoms include bilateral leg symptoms, saddle anesthesia (numbness in groin and buttocks), bowel and bladder dysfunction with urinary retention or incontinence, and sexual dysfunction. Requires emergency surgery within hours to prevent permanent paralysis and loss of bowel/bladder control.
Sciatica - When Nerves Are Compressed
Sciatica is one of the most disabling consequences of lumbar spine injuries. The sciatic nerve, the body's largest nerve, forms from L4, L5, S1, S2, and S3 nerve roots. When any of these roots are compressed, patients experience the characteristic radiating leg pain called sciatica.
Living With Sciatica
Sciatica dramatically impacts daily life and work capacity. Standing, sitting, walking, bending, and lifting all aggravate symptoms. Many patients cannot sit for more than 15-30 minutes before pain becomes unbearable, making desk jobs impossible without frequent breaks. Physical jobs involving standing, walking, or lifting become completely impossible.
Sleep disturbances are common. Patients cannot find comfortable positions. They wake repeatedly from pain when shifting position during sleep. Chronic sleep deprivation worsens pain perception and causes cognitive problems, mood changes, and reduced function.
Simple daily activities become challenging. Putting on socks and shoes, getting in and out of cars, climbing stairs, carrying groceries, playing with children - activities previously taken for granted become painful or impossible.
Treatment for Sciatica and Disc Herniations
Conservative treatment forms the first line for most disc herniations:
Medications: NSAIDs like ibuprofen or naproxen reduce inflammation. Muscle relaxants address protective muscle spasm. Neuropathic pain medications like gabapentin or pregabalin specifically target nerve pain. Oral corticosteroids provide stronger anti-inflammatory effect for acute radiculopathy.
Physical therapy: Core strengthening, postural training, manual therapy, and modalities. However, aggressive physical therapy can worsen acute disc herniations. Therapy timing and approach must be individualized.
Epidural steroid injections: Corticosteroid medication injected into the epidural space around the compressed nerve root reduces inflammation. Performed under fluoroscopic guidance for accuracy. Can provide dramatic relief lasting weeks to months. Multiple injections may be needed. Significant relief after injection confirms the diagnosis and documents which nerve root is causing symptoms.
Microdiscectomy surgery: When conservative treatment fails after 6-12 weeks, microdiscectomy removes the herniated disc fragment compressing the nerve. Success rates are high for appropriately selected patients - approximately 85-90% experience significant relief. Recovery takes 4-6 weeks for return to sedentary work, 3-6 months for physical labor. Some patients have residual symptoms or recurrent herniations.
Lumbar fusion: For unstable spine, multiple level disease, or failed prior surgery, fusion joins two or more vertebrae with bone graft and hardware. Recovery is longer than microdiscectomy, typically 3-6 months before return to work. Fusion significantly increases case values due to permanency, recovery time, and potential for adjacent segment disease.
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Spinal fractures range from stable compression fractures to unstable burst fractures threatening the spinal cord. These injuries typically result from high-energy trauma.
Types of Spinal Fractures
Compression fractures: The vertebral body collapses, usually from axial loading forces. Common in falls from height or high-speed collisions. When mild, may be stable and treated conservatively with bracing. When severe, can cause loss of height, spinal deformity, and chronic pain. Elderly individuals with osteoporosis suffer compression fractures from relatively minor trauma.
Burst fractures: Severe compression causes the vertebral body to shatter, with bone fragments potentially driven backward into the spinal canal. These are unstable fractures threatening the spinal cord. Often require surgical fixation with instrumentation to stabilize the spine and decompress neural structures.
Chance fractures: Horizontal fractures through the vertebral body, pedicles, and posterior elements, typically from flexion forces during accidents when wearing lap belts without shoulder restraints. Associated with abdominal injuries. Require surgical stabilization.
Facet fractures and dislocations: The small facet joints connecting adjacent vertebrae can fracture or dislocate during trauma. Bilateral facet dislocations are highly unstable and frequently cause spinal cord injury. Require emergency surgical reduction and fusion.
Spinal Cord Injuries
The most devastating spine injuries involve spinal cord damage. The spinal cord cannot regenerate after injury, making these injuries permanent.
Complete spinal cord injury: Total loss of motor and sensory function below the injury level. Cervical injuries cause quadriplegia. Thoracic and lumbar injuries cause paraplegia. Patients require lifetime care, assistive devices, home modifications, and attendant care. Life expectancy is reduced. Case values reach millions of dollars.
Incomplete spinal cord injury: Partial preservation of function below the injury level. Central cord syndrome, anterior cord syndrome, Brown-Sequard syndrome, and cauda equina syndrome represent different incomplete injury patterns. Prognosis varies - some patients recover significant function, while others have permanent disability.
Even "incomplete" spinal cord injuries cause devastating consequences including partial paralysis, bowel and bladder dysfunction, sexual dysfunction, chronic neuropathic pain, loss of independence, inability to work, and shortened life expectancy.
Chronic Back Pain and Failed Back Syndrome
Some spine injury victims develop chronic pain syndromes that persist despite appropriate treatment. These conditions dramatically impact quality of life and work capacity.
Chronic Mechanical Back Pain
Post-traumatic back pain can become chronic from multiple pain generators including degenerative disc disease accelerated by trauma, facet joint arthropathy from damaged cartilage, ligament laxity causing segmental instability, persistent muscle dysfunction and spasm, and sacroiliac joint dysfunction.
Chronic mechanical back pain creates activity intolerance. Patients cannot sit, stand, walk, or maintain any position for extended periods. They require frequent position changes, alternating sitting and standing. Physical work becomes impossible. Even sedentary work is limited by inability to maintain positions.
Failed Back Surgery Syndrome
Approximately 10-40% of spine surgery patients continue experiencing significant pain after surgery - a condition called failed back surgery syndrome (FBSS). Causes include inadequate decompression leaving residual nerve compression, recurrent disc herniation at the same or adjacent level, epidural fibrosis (scar tissue) around nerve roots, hardware complications, adjacent segment disease after fusion, and persistent neuropathic pain despite structural correction.
FBSS is particularly challenging to treat. Additional surgery has lower success rates. Patients often require long-term pain management including medications, injections, radiofrequency ablation, and potentially spinal cord stimulators. Many become permanently disabled.
Spinal Cord Stimulators
When conservative pain management fails, spinal cord stimulators may be implanted. These devices deliver electrical impulses masking pain signals. While they can reduce pain, they don't restore function. They require surgical implantation, have significant complication rates including infection and lead migration, and cost tens of thousands of dollars for initial implantation plus ongoing maintenance.
The need for a spinal cord stimulator documents severe, permanent pain that has failed all other treatments. This dramatically impacts case value.
Proving Causation in Spine Injury Cases
Insurance companies aggressively dispute whether accidents caused spine injuries, particularly when pre-existing degenerative changes exist on imaging.
The Pre-Existing Condition Defense
Most adults over 40 have some degenerative disc disease visible on MRI. Insurance companies obtain all prior medical records and imaging, arguing current symptoms result from pre-existing degeneration rather than accident trauma.
California's eggshell plaintiff doctrine protects against this defense. Defendants take victims as they find them. However, proving the accident aggravated rather than simply revealed pre-existing conditions requires strong evidence.
Evidence Proving Traumatic Aggravation
- Comparison imaging: Pre-accident and post-accident MRI showing new herniations or significant worsening of existing disc problems
- Asymptomatic before accident: Medical records and testimony proving you had no back problems or minimal symptoms before the accident despite degenerative changes
- Work history: Successfully performing physically demanding work before the accident proves prior degeneration was asymptomatic
- Immediate symptom onset: Documentation of back pain beginning at the accident scene or within hours
- Mechanism of injury: Biomechanical evidence that accident forces were sufficient to cause acute disc herniation
- Radiologist reports: Radiologists describing herniations as "acute" based on imaging characteristics like high-intensity zones indicating recent tears
- Physician opinions: Treating orthopedists or neurosurgeons opining that the accident caused acute injury superimposed on chronic degeneration
Biomechanical Evidence
Biomechanical engineers reconstruct accidents to calculate forces experienced by the spine. They analyze vehicle speeds and accelerations, direction and magnitude of impact forces, occupant kinematics during the collision, and comparison to known injury thresholds for disc herniation.
This testimony counters insurance arguments that accidents were too minor to cause serious injury. Research documents that disc herniations can occur in relatively low-speed collisions, particularly in people with pre-existing degeneration making discs more vulnerable.
Temporal Relationship
The timing of symptom onset relative to the accident is critical. Immediate or rapidly developing symptoms strongly support causation. However, symptoms sometimes develop over days as disc herniations progress and inflammation develops.
Documentation proving temporal relationship includes emergency department records documenting back pain complaints, early physician visits within days of accident, imaging studies obtained shortly after accident showing acute findings, and daily symptom journals tracking onset and progression.
Work Restrictions and Vocational Impact
Spine injuries frequently prevent return to previous employment, particularly physical occupations. Quantifying this economic loss requires comprehensive vocational analysis.
Functional Capacity Evaluations
Physical therapists or occupational therapists conduct standardized functional capacity evaluations measuring lifting capacity, carrying ability, push/pull strength, standing and walking tolerance, sitting tolerance, bending and twisting ability, climbing stairs and ladders, and overhead work capacity.
Results identify specific restrictions like no lifting over 10 pounds occasionally and 5 pounds frequently (sedentary work), no lifting over 20 pounds occasionally and 10 pounds frequently (light work), or inability to sit or stand for prolonged periods without position changes.
Vocational Expert Analysis
Vocational rehabilitation experts compare your functional restrictions to your previous job demands. They determine whether you can return to past work with or without accommodations, identify alternative occupations you could perform with your restrictions, calculate wage differences between past employment and available alternative work, and project lifetime earning losses.
For example, a construction worker earning $75,000 annually who can no longer perform physical labor but could potentially work in sedentary positions paying $35,000 suffers $40,000 annual lost earning capacity. Projected over a 30-year work life remaining, this represents over $1.2 million in lost earnings (present value calculation accounting for receiving money now instead of over time).
Total Disability
Some spine injuries cause complete inability to work in any capacity. Factors supporting total disability include severe chronic pain preventing sustained activity, neurological deficits precluding most work, inability to sit, stand, or maintain any position tolerably, cognitive effects from chronic pain and medications, or combination of physical restrictions and other factors like age, education, and work history limiting alternative employment options.
Total disability claims require particularly strong medical and vocational evidence given their dramatic impact on case value.
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Spine injury case values vary dramatically based on multiple factors. Understanding these factors helps evaluate settlement offers and set realistic expectations.
Economic Damages
Medical expenses: Past treatment costs including emergency care, imaging studies, physician consultations, physical therapy, injections, surgery, medications, and medical equipment. Future medical costs projected by life care planners including ongoing physician follow-up, future injections, potential additional surgeries, long-term medications, assistive devices, and home health care if needed.
Lost wages and earning capacity: Past lost income from missed work and reduced hours. Future lost earning capacity calculated by vocational experts and economists accounting for wage differences between past and available employment, lost advancement opportunities, lost benefits, and total disability if unable to work.
Non-Economic Damages
Pain and suffering: Physical pain endured from the injury. Chronic back pain lasting years deserves substantial compensation. Daily pain diaries documenting suffering strengthen these claims.
Loss of enjoyment of life: Activities you can no longer perform including recreational activities, hobbies, yard work and home maintenance, playing with children or grandchildren, intimate relations, and travel. These quality of life losses deserve significant compensation.
Emotional distress: Chronic pain causes depression, anxiety, and psychological suffering. Loss of independence and identity, especially for those who defined themselves through physical capabilities. Strain on family relationships. Mental health treatment records and testimony from family members strengthen these claims.
Factors Affecting Spine Injury Case Values
| Factor | Impact on Value |
|---|---|
| Herniation size and type | Large extrusions and sequestrations worth more than small bulges |
| Neurological deficits | Objective weakness, reflex changes significantly increase value |
| Surgical intervention | Surgery dramatically increases values - fusion more than microdiscectomy |
| Permanent restrictions | Physician-imposed work restrictions justify higher awards |
| Age of victim | Younger victims have longer period living with disability and greater lost earning capacity |
| Pre-injury earnings | Higher earners have greater economic losses |
| Physical occupation | Spine injuries have more impact when work requires physical labor |
| Clear liability | Strong liability increases settlement value and jury verdict potential |
| Available insurance | Higher policy limits allow full compensation |
Typical Settlement and Verdict Ranges
These ranges represent general guidelines. Actual values depend on specific case facts:
Lumbar strain with full recovery: $10,000 - $35,000
Disc bulge with conservative treatment: $35,000 - $85,000
Herniated disc requiring epidural injections: $85,000 - $250,000
Herniated disc with radiculopathy and neurological deficits: $200,000 - $500,000
Microdiscectomy surgery: $250,000 - $600,000
Single-level lumbar fusion: $400,000 - $1,000,000
Multi-level fusion or failed back syndrome: $750,000 - $2,500,000+
Compression fracture: $200,000 - $800,000 depending on stability and treatment
Spinal cord injury with incomplete paralysis: $2,000,000 - $10,000,000+
Complete spinal cord injury: $5,000,000 - $30,000,000+ depending on injury level and age
Why Medical Documentation Is Critical
Winning spine injury cases requires comprehensive medical evidence proving injury severity, causation, and permanency.
MRI Imaging Requirements
MRI is the gold standard for diagnosing spine injuries. Proper MRI protocols include sequences showing anatomy (T1-weighted), highlighting fluid and pathology (T2-weighted), and emphasizing edema and acute changes (STIR sequences). Axial cuts through each disc level show disc morphology, neural foramina, and nerve root compression.
Radiologist reports should specify disc herniation type, size, and location, degree of neural foraminal stenosis, nerve root compression, spinal canal narrowing, and any signal changes indicating acute injury versus chronic degeneration.
Serial MRI studies over time document progression or persistence of herniations, development of new herniations at adjacent levels, and post-surgical changes or recurrent herniations.
Physician Opinions
Orthopedic surgeons or neurosurgeons provide authoritative opinions on spine injuries. Their reports should include detailed accident history and mechanism of injury, comprehensive physical examination with neurological testing, review and interpretation of imaging studies, diagnosis with specific anatomic levels, treatment plan with medical justification, causation opinion linking accident to documented injuries, prognosis including whether symptoms are permanent, and work restrictions based on functional limitations.
Multiple examinations over time document symptom course. Consistency between examinations strengthens credibility. Documentation of treatment attempts and results proves conservative measures failed before recommending surgery.
Electrodiagnostic Testing
EMG (electromyography) and nerve conduction studies objectively document nerve damage from disc herniations or other nerve compression. Abnormal findings prove radiculopathy when insurance companies claim pain is subjective exaggeration. Denervation patterns identify which nerve roots are affected, confirming clinical and MRI findings. Follow-up studies document whether nerve damage is improving or permanent.
Our Approach to Spine Injury Cases
Spine injuries require focused legal representation from attorneys who understand the medical complexity and have trial experience proving these cases.
Focus on Orthopedic Injuries
Unlike attorneys handling any case type, we focus our practice on orthopedic and neurological injuries including spine and back trauma. This focus allows us to stay current with medical literature on spine injuries, understand evolving diagnostic and treatment approaches, maintain relationships with respected medical professionals, and develop effective litigation strategies specific to spine cases.
Comprehensive Medical Evidence
We ensure clients obtain proper diagnostic imaging including MRI if not yet performed, coordinate independent examinations by orthopedic surgeons or neurosurgeons, arrange functional capacity evaluations documenting work restrictions, obtain life care planning for chronic injuries requiring ongoing treatment, secure vocational expert analysis quantifying lost earning capacity, and utilize biomechanical experts when causation is disputed.
Trial Experience Matters
Our 25+ years includes extensive trial experience. Insurance companies evaluate attorneys' trial capabilities when making settlement offers. They know we prepare every case for trial from day one, have successfully tried complex spine injury cases, and achieve favorable verdicts when cases proceed to trial.
This trial reputation creates settlement leverage. Insurance companies make fair offers to attorneys they respect as trial lawyers while lowballing attorneys who always settle.
Contingency Fee Structure
We handle all spine injury cases on contingency fees. You pay nothing upfront and nothing if we don't recover money for you.
Our Fee Structure
29% before filing lawsuit: Lower than most attorneys who charge 33⅓% at all stages
33⅓% after filing lawsuit: Standard rate once litigation begins
40% if trial required: Reflects extensive preparation and courtroom time for trial
Calculated on net recovery: We calculate fees after deducting costs, meaning you keep more compared to attorneys calculating on gross settlement
No recovery = no fee: If we don't win, you owe nothing for attorney fees or costs
Case Investment
Properly prosecuting spine injury cases requires investment in medical evidence including obtaining complete medical records, MRI or CT imaging if not yet performed, independent medical examinations, biomechanical expert analysis for disputed causation, life care planning for permanent injuries, vocational expert evaluations, EMG/NCV testing documenting nerve damage, and deposition costs for parties and experts.
We advance all costs during your case and are only reimbursed if we recover money for you. This contingency cost advancement allows you to pursue maximum compensation without financial risk.
Common Questions About Spine Injury Cases
How long after an accident can disc herniations develop? Some herniations occur immediately from traumatic forces. Others develop progressively over days to weeks as small annular tears enlarge and nucleus material migrates through the tears. This delayed progression is medically documented and doesn't mean the injury is unrelated to the accident. Serial MRI showing progression from small protrusion to larger extrusion proves trauma initiated the degenerative cascade.
Will I definitely need surgery? No. Many disc herniations improve with conservative treatment including physical therapy, medications, and injections. Surgery is reserved for cases where conservative treatment fails, neurological deficits worsen, or symptoms remain disabling after appropriate non-surgical care. Your physicians determine medical necessity, not insurance companies.
What if I have degenerative disc disease on my MRI? Most adults over 40 have some degenerative changes. The key question is whether you were asymptomatic before the accident despite these changes. If the accident converted you from asymptomatic to symptomatic, you deserve compensation. Pre-accident medical records showing no back complaints, work history demonstrating physical capability, and immediate symptom onset after the accident all prove traumatic aggravation.
Can I sue if the accident was partially my fault? Yes. California uses pure comparative negligence. Your recovery is reduced by your percentage of fault. If you were 25% at fault and your damages are $500,000, you recover $375,000. Partial fault doesn't bar recovery.
How long will my case take? Timeline varies based on injury severity and treatment course. Cases cannot settle until reaching maximum medical improvement and understanding whether injuries are permanent. For injuries requiring surgery, this typically means 6-12 months after surgery. Complex cases with disputed liability or causation may take 2-4 years through trial. Never rush settlement before fully understanding your injuries and prognosis.
What if I can't afford treatment? Medical providers often treat accident victims on liens, meaning they defer payment until your case resolves. We can help arrange treatment with providers who work on this basis. Additionally, your health insurance may cover accident treatment, with reimbursement from settlement proceeds. Don't let cost prevent proper medical care - this harms both your health and your case.
Take Action to Protect Your Rights
If you've suffered spine or back injuries in an accident, prompt action protects both your health and legal rights.
Immediate Steps
What To Do After Spine Injuries
- Seek immediate medical evaluation: Emergency department or urgent care for assessment and documentation
- Follow treatment recommendations: Complete imaging studies, specialist referrals, and therapy as prescribed
- Document everything: Keep symptom journals, save medical records and bills, photograph accident scene and vehicles
- Avoid insurance adjuster statements: Politely decline recorded statements and don't sign medical authorizations
- Don't post on social media: Insurance companies monitor all platforms and misuse posts
- Consult an attorney immediately: Early legal involvement preserves evidence and prevents costly mistakes
- Don't accept early settlement offers: Initial offers are typically far below true case value
Why Choose Phillips Personal Injury
Located in downtown Nevada City at 305 Railroad Avenue, we've served Nevada County residents for over 25 years. Our focus on orthopedic injuries means we understand spine injury medicine, maintain relationships with respected medical professionals, and have the trial experience to maximize compensation.
Insurance companies know our reputation. They make fair settlement offers when they know we're prepared to take cases to trial if necessary. This trial readiness produces better outcomes than attorneys who settle every case regardless of adequacy.
Call (530) 265-0186 today for a free consultation. We'll review your case, explain your rights, and outline the steps necessary to maximize your recovery. No fee unless we win your case.